Drug Shows Promise for Controlling Cancer Virus
When the cancer-causing Epstein-Barr virus moves into a B-cell of the human immune system, it tricks the cell into rapidly making more copies of itself, each of which will carry the virus.
To satisfy a sudden increase in demand for more building parts, rapidly dividing host cells will chew up their insides to free up more amino acids, fats and nucleotides.
But if supplies of these materials run low, the cell will enter a suspended state called senescence and cell division will stop, freezing the advance of the virus, according to new findings from a Duke University research team that appear in Proceedings of the National Academy of Sciences. Read more at DukeToday.